TUBERAL HYPOTHALAMIC NEURONS SECRETING THE SATIETY MOLECULE NESFATIN-1 ARE CRITICALLY INVOLVED IN PARADOXICAL (REM) SLEEP HOMEOSTASIS.

Tuberal hypothalamic neurons secreting the satiety molecule Nesfatin-1 are critically involved in paradoxical (REM) sleep homeostasis.

The recently discovered Nesfatin-1 plays a role in appetite regulation as a satiety factor through hypothalamic leptin-independent mechanisms.Nesfatin-1 is co-expressed with Melanin-Concentrating Hormone (MCH) in neurons from the tuberal hypothalamic area (THA) which are recruited during sleep Denim states, especially paradoxical sleep (PS).To help

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Nonlocality without entanglement: Quantum theory and beyond

Quantum nonlocality without entanglement (Q-NWE) captures Course a pied - Accessoires - Hydratation - Reservoir nonlocal behavior of multipartite product states as they may entail global operation for optimal decoding of the classical information encoded in the state ensemble that allows local preparation.In this Rapid Communication we show that th

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